HHV Solutions

Hit-to-Lead Generation Solutions for HHV Drug Discovery

At CD BioSciences, we specialize in transforming early screening hits into validated, structurally optimized, and biologically characterized lead compounds specifically targeting human herpesviruses (HHVs). Our Hit-to-Lead (H2L) Generation Solutions are built upon a robust understanding of the complex biology of alpha-, beta-, and gamma-herpesviruses, with specialized capabilities in viral replication, latency, reactivation, and host-pathogen interaction.

Whether you're pursuing novel targets such as viral helicases, proteases, or latency-associated proteins like LANA, or repurposing broad-spectrum antivirals with anti-HHV activity, CD BioSciences offers integrated preclinical H2L services tailored to HHV biology and druggability.

Our HHV-Focused Hit-to-Lead Workflow

We provide a structured yet flexible workflow, optimized for HHV-specific antiviral drug discovery:

Hit Prioritization & Characterization

  • Mechanism-based triage: We prioritize hits based on relevance to HHV targets (e.g., viral polymerases, immediate-early genes, tegument proteins).
  • Assay reproducibility: All hits undergo retesting in orthogonal cell-based assays, including plaque reduction, cytopathic effect (CPE), and HHV-specific reporter systems.
  • Cytotoxicity profiling: Evaluation of CC50 and selectivity index (SI) in host-relevant cell lines (e.g., Vero, HEK293, BCBL-1).

Structure-Activity Relationship (SAR) Exploration

  • Medicinal chemistry design: SAR hypotheses are informed by the structure and physicochemical properties of prioritized hits.
  • Analog library synthesis: Rapid synthesis and evaluation of analogs with modified scaffolds, substitutions, or linker optimizations.
  • Activity refinement: Continuous in vitro evaluation against HHV strains (e.g., HSV-1, HSV-2, HCMV, EBV, KSHV) to monitor potency improvements.

Mechanism of Action (MoA) Elucidation

  • Time-of-addition studies: To identify early vs. late viral lifecycle intervention points.
  • Resistance profiling: Selection of resistant HHV strains under drug pressure, followed by sequencing of viral genes.
  • Target validation: Combined use of genetic knockouts, overexpression, or CRISPR interference with compound treatment.

Lead Compound Selection Criteria

  • Potency & selectivity: IC50 < 1 μM and SI > 100 preferred for nomination.
  • Drug-likeness: Compliance with Lipinski's Rule of Five, favorable logP, PSA, and molecular weight.
  • ADME screening: In vitro metabolic stability (liver microsomes), permeability (Caco-2), plasma protein binding, and solubility profiling.

Key Advantages of CD BioSciences' HHV H2L Services

Deep HHV Expertise

We maintain a virus-specific focus across all herpesvirus subfamilies—offering specialized knowledge in latency-associated gene expression (e.g., LANA in KSHV, EBNA1 in EBV), strain-specific tropism, and immune evasion.

Proprietary Tools & Assays

  • HHV reporter viruses: For rapid antiviral screening.
  • Latency reactivation assays: To test for efficacy in latent or reactivating virus contexts.
  • Custom HHV cell models: Including primary neuronal and B cell latency models.

Seamless Integration with Upstream & Downstream Services

Our Hit-to-Lead generation is fully embedded within a larger antiviral discovery pipeline:

  • Upstream: Hit identification via high-throughput screening or in silico docking.
  • Downstream: Preclinical candidate nomination, IND-enabling studies, and PK/PD modeling.

Accelerated Timelines

With internal chemistry, virology, and assay teams, we rapidly iterate SAR cycles and deliver qualified lead compounds with defined MoA and pre-IND data packages.

Application Areas

Our HHV-specific hit-to-lead services have been applied in the discovery of antivirals targeting:

  • Herpes Simplex Viruses (HSV-1/2): Viral helicase-primase inhibitors, entry blockers, latency reactivators.
  • Varicella-Zoster Virus (VZV): DNA polymerase and capsid assembly inhibitors.
  • Human Cytomegalovirus (HCMV): UL97 kinase and terminase complex inhibitors.
  • Epstein-Barr Virus (EBV): LMP1/EBNA1 disruptors, latency reversing agents.
  • Kaposi's Sarcoma-associated Herpesvirus (KSHV): LANA-targeted small molecules, K-Rta activators.

Collaborative Project Models

We offer flexible business and collaboration models:

  • Fee-for-Service (FFS): Full execution of your hit-to-lead campaign with regular updates.
  • FTE-based: Embedded scientist model for high-volume iterative SAR work.
  • Milestone-based: Shared-risk models tied to lead optimization goals.

Typical Deliverables

By the end of a hit-to-lead project, you will receive:

  • Prioritized lead series with SAR documentation.
  • Confirmed MoA and antiviral profiling data.
  • Drug-likeness and early ADME characterization.
  • Recommendations for lead optimization and IND strategy.

Why Choose CD BioSciences?

  • HHV specialization: We are the only platform with exclusive focus on HHV biology and preclinical drug development.
  • Multidisciplinary integration: In-house medicinal chemistry, virology, molecular biology, and bioinformatics expertise.
  • Tailored solutions: Custom design of screening and SAR strategies for your target class or indication.
  • Regulatory awareness: We generate data packages aligned with preclinical requirements for US and EU regulatory filings.

Contact us to initiate your HHV-focused hit-to-lead project. Whether you are screening broad antiviral libraries or fine-tuning leads against latent reservoirs, CD BioSciences is your expert partner in HHV drug discovery.

References

  1. De Clercq E, Li G. Approved Antiviral Drugs over the Past 50 Years. Clin Microbiol Rev. 2016;29(3):695–747. doi:10.1128/CMR.00102-15
  2. Prichard MN, Kern ER. The search for new therapies for human cytomegalovirus infections. Virus Res. 2011;157(2):212–221.
  3. Wyles DL, Marcellin P, et al. Challenges in Drug Development for Latent Viral Infections. Nat Rev Drug Discov. 2020;19(12):878–900.
  4. Liu Y, et al. Identification of a Small-Molecule Inhibitor Targeting Kaposi's Sarcoma-Associated Herpesvirus LANA. J Virol. 2022;96(4):e0176121.
  5. Andrei G, Snoeck R. Advances in the treatment of alpha-herpesvirus infections. Curr Opin Infect Dis. 2013;26(3):231–239.

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